H J Fr Ophtalmol. 2005 Jun;28(6):642-5. Links
[Recovery of sickle cell disease ischemic maculopathy after
erythropheresis: a clinical case study.][Article in French]
Leveziel N, Kirsch O, Lautier-Frau M, Driss F, Offret H, Labetoulle
M.
Service d'Ophtalmologie, CHU du Kremlin-Bicetre, Le Kremlin-Bicetre,
Paris.

The authors report a case of a young patient with a recent decrease in
unilateral vision. He had homozygote sickle cell disease with multiple
general complications. Fundus examination was normal apart from a mild
alteration of the macular reflect in the left eye, but fluorescein
angiography showed multiple arteriolar macular occlusions, explaining
the decrease in vision in the left eye. After erythropheresis, vision
acuity improved and fluorescein angiography showed reperfusion. This
case suggests that transfusional exchange may improve acute macular
ischemia secondary to sickle cell disease.

PMID: 16141931 [PubMed - indexed for MEDLINE]



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When one goes to altitude one presents with increased red blood cell
production which is accompanied by .. hyperviscous .. blood.
Coincidentally .. again .. they acquire .. **monocular vision
loss** .. coincidentally .. again .. the SAME .. 'elevations' .. which
trigger / manifest .. multiple sclerosis at a HIGH .. rate / ten times
the rate.
Coincidentally .. monocular vision loss / optic neuritis ..
PRESENTS .. multiple sclerosis.

So can .. vision problems BE .. a MARKER .. for .. **hyperviscous
blood** .. ? .. since multiple sclerosis presents with very high rate
of hyperviscosity .. ?

Which would .. warrant .. as evidenced .. treatment WITH ..
bloodletting / phlebotomy.

Any other intervention would / should be considered .. criminal.

Since it is KNOWN to treat AND .. **cure** .. vision loss due to
monocular .. vision .. loss / optic neuritis.

Stroke. 1990 Jan;21(1):34-9. Links
Transient monocular visual loss patterns and associated vascular
abnormalities.Bruno A, Corbett JJ, Biller J, Adams HP Jr, Qualls C.
Neurology Service, Veterans Administration Medical Center,
Albuquerque, NM 87108.

To determine if certain transient monocular visual loss patterns
predict the associated vascular abnormalities, we prospectively
evaluated 100 consecutive patients. Each patient had hematologic
tests, a carotid artery study (arteriography in 74, duplex
ultrasonography in the remaining 26), and an ophthalmologic
examination. Patients with altitudinal or lateralized transient
monocular visual loss were more likely to have carotid artery
stenosis, carotid artery ulceration, cardiac sources of emboli, or
visible retinal emboli than patients with other visual loss patterns.
Our findings suggest that altitudinal or lateralized transient
monocular visual loss is primarily caused by embolism but that other
visual loss patterns are usually caused by nonembolic mechanisms.

PMID: 2300989 [PubMed - indexed for MEDLINE]


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Jesus Was A Vegetarian!
http://jesuswasavegetarian.7h.com


Man Is A Herbivore!
http://tinyurl.com/a3cc3


DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk

I like Valvoline. Good viscosity.

Optic neuritis presents with monocular vision loss.
Monocular vision loss has been shown to be treated with phlebotomy /
bloodletting.
Monocular vision loss is caused by the hyperviscosity of the blood.
Multiple sclerosis presents with .. optic neuritis .. AND is
accompanied by .. hyperviscosity.

Coincidence .. of .. course.

<<snip> due to the great viscosity of blood
<<snip>
J Neurol Sci. 2007 Apr 25; [Epub ahead of print] Links
Isolated monocular visual loss as an initial manifestation of
polycythemia vera.Ahn BY, Choi KD, Choi YJ, Jea SY, Lee JE.
Department of Neurology, Pusan National University Hospital, Pusan
National University School of Medicine and Medical Research
Institute,
Republic of Korea.


A 25-year-old man developed prolonged loss of vision in the left eye.
Examination revealed that visual acuity was 20/20 in the right eye
and
10/20 in the left eye, with a left relative afferent pupillary
defect.
Fundoscopy showed multiple cotton wool spots in the left whole retina
with normal optic disc. Fluorescein angiography (FA) revealed
markedly
delayed arterial, venous and recirculation time in the left eye
without retinal arterial or venous occlusion. Bone marrow aspirate
confirmed polycythemia vera. After the patient underwent phlebotomy,
his visual acuity markedly improved and cotton wool spots in the
retina disappeared. On follow-up FA, delayed arterial and venous
filling, and recirculation time also became normalized. This case
suggests that ischemic damage of the retina due to the great
viscosity
of blood may be a possible mechanism of monocular visual loss in
polycythemia vera. Clinicians should be aware that isolated monocular
visual loss may be an initial manifestation of polycythemia vera,
since if untreated, polycythemia vera carries a high risk of
permanent
complications due to intravascular thrombosis.


PMID: 17466335 [PubMed - as supplied by publisher]
Rev Neurol Dis. 2006 Spring;3(2):45-56. Links
The natural historyof optic neuritis.Atkins EJ, Biousse V, Newman NJ.
Department of Neurology, University of Saskatchewan, Saskatoon, SK,
Canada.

Optic neuritis is a common cause of visual loss in young patients,
typically presenting with painful monocular visual loss and decreased
color vision. Visual function generally spontaneously improves over
weeks, and 95% of patients return to visual acuity of at least 20/40
within 12 months. The initial magnetic resonance imaging (MRI) helps
stratify the risk of multiple sclerosis (MS) in patients with acute
isolated optic neuritis. In the Optic Neuritis Treatment Trial, the 10-
year risk of MS in the group of patients with at least one MRI T2
lesion was 56%, whereas the 10-year risk with a normal baseline MRI
was 22%. A normal MRI in concert with painless optic neuritis, severe
optic nerve head edema, peripapillary hemorrhages, or a macular star
defines a very low MS risk subgroup. High-dose steroids hasten the
rate, but not the final extent, of visual recovery in optic neuritis,
and the decision to use this therapy is individualized. Interferon
beta-1a therapy should be considered in selected high-risk patients.

PMID: 16819420 [PubMed - in process]


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Jesus Was A Vegetarian!
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Man Is A Herbivore!
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DEAD PEOPLE WALKING
http://tinyurl.com/zk9fk